At the point of prescription

Right drug, right dose — pre-emptively.

Adverse drug reactions and trial-and-error dosing are avoidable when genotype is known before the prescription is written. Test once, use for life.

Why PGx is the strongest wedge

Six properties that make it work.

Test once, use for life

Cost amortises across every subsequent prescription the patient ever receives.

Deterministic mapping

Diplotype to phenotype to guidance runs through versioned consortium tables. No inference in the path.

Concrete action

A dose adjustment or an alternative agent — not a risk score to interpret.

Measurable benefit

Avoided adverse events are the strongest procurement argument available.

Established guidelines

CPIC-aligned mappings provide a defensible, citable evidence base.

Cross-module value

Applies in diabetes, oncology, cardiology and psychiatry alike.

How it works

From diplotype to dosing guidance.

01
Diplotype calling

From the molecular layer, with assay limitations recorded explicitly rather than assumed away.

02
Phenotype translation

Deterministic mapping using versioned consortium tables — the language model is not in this path.

03
Surfaced at prescription

Guidance appears at the moment the drug is chosen, not in a report the prescriber may never open.

04
Interactions checked together

Drug–drug, drug–gene and drug–nutrient, because in chronic disease they co-occur.

05
Stored in the twin

A genotype tested once is reused at every prescribing event for the rest of the patient’s life.

Deployment

Interoperable with what you already run.

EHR integrationResults and guidance delivered via HL7 FHIR into existing workflows; SMART on FHIR launch where supported.
Pharmacy systemsInteroperable output for dispensing systems and pharmacy networks.
Panel or genomeWorks from a targeted PGx panel or from existing whole-genome data.
GovernanceEvery recommendation evidence-linked, reviewed and signed — never auto-applied.
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Anchor sites get early capability and a real say in the roadmap.